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- W2000201026 endingPage "611" @default.
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- W2000201026 abstract "Electrolytic lesions of the medial core of the nucleus accumbens (NAc) in male Long-Evans rats increased spontaneous locomotion, enhanced the locomotor stimulating effect of acute 5.0 mg/kg cocaine, enhanced the development and subsequent expression of locomotor sensitization produced by repeated injections of 0.4 mg/kg nicotine but not 7.5 mg/kg cocaine, and enhanced the expression of conditioned locomotion. Given that 6-hydroxydopamine lesions of the NAc typically have effects on locomotor-related processes that are opposite of those produced by electrolytic and excitotoxic lesions, these data are consistent with a hypothesis that the NAc output, especially from the core, inhibits a variety of such processes and that the DA input to the NAc enhances these processes by inhibiting this inhibitory output." @default.
- W2000201026 created "2016-06-24" @default.
- W2000201026 creator A5000852396 @default.
- W2000201026 creator A5025829731 @default.
- W2000201026 date "2006-01-01" @default.
- W2000201026 modified "2023-09-24" @default.
- W2000201026 title "Electrolytic lesions of the nucleus accumbens enhance locomotor sensitization to nicotine in rats." @default.
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- W2000201026 doi "https://doi.org/10.1037/0735-7044.120.3.600" @default.
- W2000201026 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16768612" @default.
- W2000201026 hasPublicationYear "2006" @default.
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