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• W2000219965 abstract "Hypertension is the most common chronic disease and is the leading risk factor for death caused by stroke, myocardial infarction, and end-stage renal failure. The critical importance of excess salt intake in the pathogenesis of hypertension is widely recognized. However, the molecular mechanisms underlying salt-sensitive hypertension remain obscure. Recent studies using selective Na(+)/Ca(2+) exchanger (NCX) inhibitors and genetically engineered mice provide compelling evidence that salt-sensitive hypertension is triggered by Ca(2+) entry through NCX type 1 (NCX1) in arterial smooth muscle. Cardiotonic steroids, such as endogenous ouabain, which may contribute to the pathogenesis of salt-sensitive hypertension, seem to be necessary for NCX1-mediated hypertension. These findings have enabled us to explain how high salt intake leads to hypertension and further to describe the potential of vascular NCX1 as a new therapeutic or diagnostic target for salt-sensitive hypertension." @default.
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• W2000219965 date "2005-11-01" @default.
• W2000219965 modified "2023-10-02" @default.
• W2000219965 title "Salt-Sensitive Hypertension, Na+/Ca2+ Exchanger, and Vascular Smooth Muscle" @default.
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• W2000219965 doi "https://doi.org/10.1016/j.tcm.2005.08.004" @default.
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