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• W2000291134 startingPage "1543" @default.
• W2000291134 abstract "Adenosine triphosphate (ATP)-sensitive potassium (K(ATP)) channels are activated by various metabolic stresses, including hypoxia. The substantia nigra pars reticulata (SNr), the area with the highest expression of K(ATP) channels in the brain, plays a pivotal role in the control of seizures. Mutant mice lacking the Kir6.2 subunit of K(ATP) channels [knockout (KO) mice] were susceptible to generalized seizures after brief hypoxia. In normal mice, SNr neuron activity was inactivated during hypoxia by the opening of the postsynaptic K(ATP) channels, whereas in KO mice, the activity of these neurons was enhanced. K(ATP) channels exert a depressant effect on SNr neuronal activity during hypoxia and may be involved in the nigral protection mechanism against generalized seizures." @default.
• W2000291134 created "2016-06-24" @default.
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• W2000291134 date "2001-05-25" @default.
• W2000291134 modified "2023-10-14" @default.
• W2000291134 title "Protective Role of ATP-Sensitive Potassium Channels in Hypoxia-Induced Generalized Seizure" @default.
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• W2000291134 doi "https://doi.org/10.1126/science.1059829" @default.
• W2000291134 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11375491" @default.
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