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- W2000303712 abstract "The question of how Bax is activated during apoptosis to perform its role in permeabilization of mitochondrial membranes is intriguing for investigators in the wide field of cell death research. In their paper published in the Biochemical Journal in 2006, Capano and Crompton presented their discovery that simulated ischaemia causes rapid activation of AMPK (AMP-activated protein kinase) which phosphorylates and activates p38 MAPK (mitogen-activated protein kinase) leading to Bax activation and translocation to mitochondria in isolated cardiac myocytes. This was the first report on the molecular mechanism of Bax activation and migration during ischaemia-induced apoptosis in cardiomyocytes." @default.
- W2000303712 created "2016-06-24" @default.
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- W2000303712 date "2008-05-14" @default.
- W2000303712 modified "2023-09-25" @default.
- W2000303712 title "AMPK, MAPK and Bax in the heart: some questions answered" @default.
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- W2000303712 doi "https://doi.org/10.1042/bj20080712" @default.
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