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- W2000346175 abstract "The nutritional needs of developing young place a large energetic demand on lactating females. In this paper some of the mechanisms through which lactating rats meet these demands are described. Emphasis is placed on the mechanisms that drive increased food intake in suckled rats that are independent of milk delivery and negative energy balance. Evidence is presented to suggest that prolactin (PRL), released from the pituitary in response to suckling stimulation, acts centrally to stimulate food intake during lactation. Brain areas in which PRL may act to stimulate food intake as well as its interactions with other hormones and neuropeptides involved in energy balance are discussed." @default.
- W2000346175 created "2016-06-24" @default.
- W2000346175 creator A5088499376 @default.
- W2000346175 date "2007-07-01" @default.
- W2000346175 modified "2023-10-02" @default.
- W2000346175 title "Prolactin and the hyperphagia of lactation" @default.
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- W2000346175 doi "https://doi.org/10.1016/j.physbeh.2007.04.015" @default.
- W2000346175 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17498759" @default.
- W2000346175 hasPublicationYear "2007" @default.
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