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- W2000426341 endingPage "591" @default.
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- W2000426341 abstract "Cerebrovascular dysfunction significantly contributes to the clinical presentation and pathoetiology of Alzheimer's disease (AD). Deposition and aggregation of β-amyloid (Aβ) within vascular smooth muscle cells leads to inflammation, oxidative stress, impaired vasorelaxation, and disruption of blood-brain barrier integrity. Midlife vascular risk factors, such as hypertension, cardiovascular disease, diabetes, and dyslipidemia, increase the relative risk for AD. These comorbidities are all characterized by low and/or dysfunctional high-density lipoproteins (HDL), which itself is a risk factor for AD. HDL performs a wide variety of critical functions in the periphery and CNS. In addition to lipid transport, HDL regulates vascular health via mediating vasorelaxation, inflammation, and oxidative stress and promotes endothelial cell survival and integrity. Here, we summarize clinical and preclinical data examining the involvement of HDL, originating from the circulation and from within the CNS, on AD and hypothesize potential synergistic actions between the two lipoprotein pools." @default.
- W2000426341 created "2016-06-24" @default.
- W2000426341 creator A5059433744 @default.
- W2000426341 creator A5065510480 @default.
- W2000426341 creator A5091130668 @default.
- W2000426341 date "2014-04-01" @default.
- W2000426341 modified "2023-10-14" @default.
- W2000426341 title "High-Density Lipoproteins and Cerebrovascular Integrity in Alzheimer’s Disease" @default.
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