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- W2000451365 abstract "Cholera toxin (CTX)-stimulated synthesis of adenosine 3′, 5′ monophosphate (cyclic AMP) enhances the rate of wound closure of a 7-mm-diameter corneal epithelial defect following heptanol debridement but not after mechanical scraping. Pretreatment with topical epinephrine or topical timolol does not alter the rate of wound closure following either trauma. Corneal glycogen levels decrease after heptanol debridement but glycogenosis is not influenced by compounds which increase intracellular cyclic AMP levels. We postulate that cyclic AMP increases the rate of wound closure towards a maximum; however, the first messenger for activating the pathway is not a catecholamine and the mechanism for enhancing closure is not via mobilization of glycogen stores." @default.
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- W2000451365 date "1981-01-01" @default.
- W2000451365 modified "2023-09-23" @default.
- W2000451365 title "Characterization of cyclic AMP-mediated wound closure of the rabbit corneal epithelium" @default.
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- W2000451365 doi "https://doi.org/10.3109/02713688109001848" @default.
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