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- W2000483769 endingPage "1127" @default.
- W2000483769 startingPage "1119" @default.
- W2000483769 abstract "Amyloid formation by the neuropancreatic hormone, islet amyloid polypeptide (IAPP or amylin), one of the most amyloidogenic sequences known, leads to islet amyloidosis in type 2 diabetes and to islet transplant failure. Under normal conditions, IAPP plays a role in the maintenance of energy homeostasis by regulating several metabolic parameters, such as satiety, blood glucose levels, adiposity and body weight. The mechanisms of IAPP amyloid formation, the nature of IAPP toxic species and the cellular pathways that lead to pancreatic β‐cell toxicity are not well characterized. Several mechanisms of toxicity, including receptor and non‐receptor‐mediated events, have been proposed. Analogs of IAPP have been approved for the treatment of diabetes and are under investigation for the treatment of obesity." @default.
- W2000483769 created "2016-06-24" @default.
- W2000483769 creator A5007603996 @default.
- W2000483769 creator A5065857864 @default.
- W2000483769 date "2013-01-18" @default.
- W2000483769 modified "2023-10-11" @default.
- W2000483769 title "Mechanisms of islet amyloidosis toxicity in type 2 diabetes" @default.
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