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- W2000603611 abstract "The contributions of (±)-a-amino-3-hydroxy-5-methylisoxazole-4-propionic acid (AMPA) and g-aminobutyric acid (GABAA) receptors in the induction of long-term potentiation (LTP) have been studied in the CA1 region of the rat hippocampus. The results suggest that: (1) in physiological conditions, AMPARs are necessary for the induction of N-methyl-d-aspartate receptor (NMDAR)-dependent LTP since LTP cannot be elicited in the presence of the AMPAR antagonist, 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX). Although a NMDAR-dependent LTP occurs in the presence of a GABAA antagonist and high concentrations of divalents cations, blockade of AMPARs leads to a voltage-dependent calcium channels (VDCC)-dependent LTP since its induction is blocked by nifedipine and not by APV. (2) The bicarbonate-induced GABAA receptor-mediated depolarizing response is not necessary in the induction of NMDAR-dependent or VDCC-dependent LTP since induction of these two types of LTP were not blocked by acetazolamide or in a nominally bicarbonate-free solution." @default.
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- W2000603611 date "1997-11-01" @default.
- W2000603611 modified "2023-09-25" @default.
- W2000603611 title "Biochemistry of wasp neurotoxin and its physiological effect on insect neurons" @default.
- W2000603611 doi "https://doi.org/10.1016/s0304-3940(97)90097-5" @default.
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