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- W2000608082 abstract "Background: The pathophysiology of sepsis is incompletely understood. Impaired bioavailability of L-arginine, the substrate for NO synthesis, is linked to sepsis severity, and plasma arginase has been linked to hypoargininemia in other disease states. Circulating neutrophils are increased in sepsis and constitutively express arginase. We investigated whether plasma arginase activity is increased in human sepsis and whether this is associated with neutrophil numbers and activation." @default.
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- W2000608082 date "2014-01-01" @default.
- W2000608082 modified "2023-09-29" @default.
- W2000608082 title "Increased plasma arginase activity in human sepsis: association with increased circulating neutrophils" @default.
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- W2000608082 doi "https://doi.org/10.1515/cclm-2013-0698" @default.
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