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• W2000660334 abstract "Forty Wistar rats were divided into 5 groups, including the control group, the acute pancreatitis group (AP group, induced by intraperitoneal injections of caerulein), and the AP group treated with baicalin, the AP group treated with LPS, and the AP group treated with LPS and baicalin. Pathological damage of pancreatic tissue was scored with hematoxylin and eosin (HE) staining. The mRNA expression of TNF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E3><mml:mi>α</mml:mi></mml:math>was measured with semiquantitative RT-PCR, and activation of NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E4><mml:mi>κ</mml:mi></mml:math>B was detected with flow cytometry assay. It was shown in the results that the expression of TNF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E5><mml:mi>α</mml:mi></mml:math>mRNA, activation of NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E6><mml:mi>κ</mml:mi></mml:math>B, and pathological score of AP group were all obviously higher than those of control group (<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E7><mml:mrow><mml:mi>P</mml:mi><mml:mo><</mml:mo><mml:mn>.01</mml:mn></mml:mrow></mml:math>). In AP group treated with LPS, further rise of these values were observed (<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E8><mml:mrow><mml:mi>P</mml:mi><mml:mo><</mml:mo><mml:mn>.01</mml:mn></mml:mrow></mml:math>). In the AP group treated with baicalin, activation of NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E9><mml:mi>κ</mml:mi></mml:math>B decreased (<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E10><mml:mrow><mml:mi>P</mml:mi><mml:mo><</mml:mo><mml:mn>.05</mml:mn></mml:mrow></mml:math>), and expression of TNF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E11><mml:mi>α</mml:mi></mml:math>mRNA also obviously decreased (<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E12><mml:mrow><mml:mi>P</mml:mi><mml:mo><</mml:mo><mml:mn>.01</mml:mn></mml:mrow></mml:math>), while pancreatic pathological damage was alleviated at the same time (<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E13><mml:mrow><mml:mi>P</mml:mi><mml:mo><</mml:mo><mml:mn>.01</mml:mn></mml:mrow></mml:math>); similar results were observed in AP group treated with LPS and baicalin (<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E14><mml:mrow><mml:mi>P</mml:mi><mml:mo><</mml:mo><mml:mn>.01</mml:mn></mml:mrow></mml:math>), which indicated that baicalin might be applied to inhibit NF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E15><mml:mi>κ</mml:mi></mml:math>B activating and TNF-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E16><mml:mi>α</mml:mi></mml:math>expressing so as to treat AP." @default.
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• W2000660334 title "Adjusting Effects of Baicalin for Nuclear Factor-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E1><mml:mi>κ</mml:mi></mml:math>B and Tumor Necrosis Factor-<mml:math xmlns:mml=http://www.w3.org/1998/Math/MathML id=E2><mml:mi>α</mml:mi></mml:math>on Rats With Caerulein-Induced Acute Pancreatitis" @default.
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