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- W2000668716 abstract "In the present study, acute effects of brain-derived neurotrophic factor (BDNF) on intracellular calcium concentration ([Ca2+]i) in cultured visual cortical neurons were investigated. We examined specifically whether pharmacological blockade of glutamatergic receptors interfered with effects of BDNF on [Ca2+]i. We found that blockade of N-methyl-D-aspartate (NMDA) receptors significantly reduced the number of BDNF-responsive neurons, while inhibition of metabotropic glutamate receptors (mGluR) completely prevented the effect of BDNF. By contrast, blockade of alpha-amino-3-hydroxy-5-methyl-4-isoxasole propionic acid (AMPA) receptors did not affect the BDNF-induced increase of [Ca2+]i. Our results thus suggest that glutamate-mediated excitatory pathways are involved in the BDNF-induced rise of [Ca2+]i in visual cortical neurons, and that both mGluR and NMDA receptors play a critical role in this event." @default.
- W2000668716 created "2016-06-24" @default.
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- W2000668716 date "2005-06-01" @default.
- W2000668716 modified "2023-09-24" @default.
- W2000668716 title "Contribution of glutamate receptors to brain-derived neurotrophic factor-induced elevation of intracellular Ca2+levels" @default.
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- W2000668716 doi "https://doi.org/10.1097/00001756-200506210-00019" @default.
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