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• W2000676236 endingPage "11" @default.
• W2000676236 startingPage "1" @default.
• W2000676236 abstract "Persistent infection with high-risk HPV is the etiologic agent associated with the development of cervical cancer (CC) development. However, environmental, social, epidemiological, genetic, and host factors may have a joint influence on the risk of disease progression. Cervical lesions caused by HPV infection can be removed naturally by the host immune response and only a small percentage may progress to cancer; thus, the immune response is essential for the control of precursor lesions and CC. We present a review of recent research on the molecular mechanisms that allow HPV-infected cells to evade immune surveillance and potential targets of molecular therapy to inhibit tumor immune escape." @default.
• W2000676236 created "2016-06-24" @default.
• W2000676236 creator A5058186142 @default.
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• W2000676236 creator A5086556136 @default.
• W2000676236 date "2012-01-01" @default.
• W2000676236 modified "2023-10-18" @default.
• W2000676236 title "Molecular Mechanism and Potential Targets for Blocking HPV-Induced Lesion Development" @default.
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• W2000676236 doi "https://doi.org/10.1155/2012/278312" @default.

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