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- W2000723048 abstract "The ability of muscles to regenerate successfully following damage diminishes with age and this appears to be a major contributor to the development of muscle weakness and physical frailty. Successful muscle regeneration is dependent on appropriate reinnervation of regenerating muscle. Age-related changes in the interactions between nerve and muscle are poorly understood but may play a major role in the defective regeneration. During aging there is defective redox homeostasis and an accumulation of oxidative damage in nerve and muscle that may contribute to defective regeneration. The aim of this review is to summarise the evidence that abnormal reactive oxygen species (ROS) generation in nerve and/or muscle may be responsible for the defective regeneration that contributes to the degeneration of skeletal muscle observed during aging. Identifying the importance of ROS generation in skeletal muscle during aging could have fundamental implications for interventions to prevent muscle degeneration and treatments to reverse the age-related decline in muscle mass and function." @default.
- W2000723048 created "2016-06-24" @default.
- W2000723048 creator A5054346772 @default.
- W2000723048 creator A5067840753 @default.
- W2000723048 date "2013-12-01" @default.
- W2000723048 modified "2023-09-29" @default.
- W2000723048 title "Role of reactive oxygen species in the defective regeneration seen in aging muscle" @default.
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- W2000723048 doi "https://doi.org/10.1016/j.freeradbiomed.2013.07.008" @default.
- W2000723048 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3859734" @default.
- W2000723048 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23851030" @default.
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