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• W2000736774 endingPage "13" @default.
• W2000736774 startingPage "1" @default.
• W2000736774 abstract "Growing evidence over the last few years suggests a central role of type I IFN pathway in the pathogenesis of systemic autoimmune disorders. Data from clinical and genetic studies in patients with systemic lupus erythematosus (SLE) and lupus-prone mouse models, indicates that the type I interferon system may play a pivotal role in the pathogenesis of several lupus and associated clinical features, such as nephritis, neuropsychiatric and cutaneous lupus, premature atherosclerosis as well as lupus-specific autoantibodies particularly against ribonucleoproteins. In the current paper, our aim is to summarize the latest findings supporting the association of type I IFN pathway with specific clinical manifestations in the setting of SLE providing insights on the potential use of type I IFN as a therapeutic target." @default.
• W2000736774 created "2016-06-24" @default.
• W2000736774 creator A5000325687 @default.
• W2000736774 creator A5021706761 @default.
• W2000736774 creator A5033359205 @default.
• W2000736774 date "2011-01-01" @default.
• W2000736774 modified "2023-10-16" @default.
• W2000736774 title "Activation of Type I Interferon Pathway in Systemic Lupus Erythematosus: Association with Distinct Clinical Phenotypes" @default.
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