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• W2000768439 abstract "Obesity can be a cause of secondary hypogonadism. This article describes a male patient with infertility in the setting of obesity, who was successfully treated with the aromatase inhibitor anastrozole. The authors illustrate the connections between adiposity, aromatase expression, testosterone and estradiol levels, and the suppression of gonadotropin release and spermatogenesis. Background A 29-year-old man presented to a clinic with infertility and hypogonadism in the setting of morbid obesity. On presentation, he had notable gynecomastia and a low testicular volume. The patient's weight was 154 kg and his height was 168 cm (BMI 54.5 kg/m2). Before referral to the clinic, the patient had been treated with testosterone therapy for 4 months for hypogonadism. This treatment had caused his initially low sperm concentration to fall to undetectable levels. Investigations Measurement of reproductive hormone levels, pituitary MRI, and semen analysis. Diagnosis Infertility secondary to hypogonadotropic hypogonadism and an elevated estrogen:testosterone ratio. Management Treatment with an aromatase inhibitor, anastrozole, led to normalization of the patient's testosterone, luteinizing hormone and follicle-stimulating hormone levels, suppression of serum estradiol levels, and to normalization of spermatogenesis and fertility." @default.
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• W2000768439 date "2008-06-03" @default.
• W2000768439 modified "2023-09-30" @default.
• W2000768439 title "Treatment of male infertility secondary to morbid obesity" @default.
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• W2000768439 doi "https://doi.org/10.1038/ncpendmet0844" @default.
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