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Matches in SemOpenAlex for { <https://semopenalex.org/work/W2000791632> ?p ?o ?g. }

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• W2000791632 abstract "Although it has been postulated that inflammatory cells cause the bronchial hyper-responsiveness which is diagnostic of asthma [1], until recently there has been little direct evidence of such a link. We have recently shown that calcium ionophore-activated human neutrophils and eosinophils can induce a state of human airway hyperresponsiveness in vitro [2]. In this study we have shown that the anti-inflammatory agent nedocromil sodium, 10-7m, inhibited the hyperresponsiveness induced by products released from ionophore activated neutrophils but did not inhibit the release of leukotriene B4 from the same cells. Neutrophil-induced bronchial hyperresponsiveness was also inhibited by pre-treatment of the bronchial tissues with a thromboxane A2 and prostaglandin receptor antagonist, GR32191,10-7m. These findings indicate that cyclooxygenase products are involved in bronchial hyperresponsiveness induced by inflammatory cell products in vitro and that their release can be inhibited by nedocromil sodium." @default.
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• W2000791632 date "1993-04-01" @default.
• W2000791632 modified "2023-09-27" @default.
• W2000791632 title "Neutrophil-induced human bronchial hyper responsiveness in vitro-pharmacological modulation" @default.
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• W2000791632 doi "https://doi.org/10.1111/j.1365-2222.1993.tb00318.x" @default.
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