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- W2000860057 endingPage "738" @default.
- W2000860057 startingPage "732" @default.
- W2000860057 abstract "The use of erythropoietin (EPO) has revolutionized the treatment of anaemia associated with many conditions including chronic kidney disease (CKD). However, little is known of the cellular impact of EPO on the uraemic heart. The discovery that the EPO receptor (EPOR) is also expressed on non-haematopoietic cells including cardiomyocytes highlights a role of EPO beyond haematopoiesis. Animal models of heart failure have shown EPO can potentially reverse cardiac remodelling and improve myocardial function. Damage to the kidney, during uraemia, results in a decreased EPO production, which may render the uraemic heart more susceptible to damage and heart failure. Here we review current data on the cellular actions of EPO in models of left ventricular hypertrophy and heart failure and highlight parallels with the uraemic heart." @default.
- W2000860057 created "2016-06-24" @default.
- W2000860057 creator A5015885243 @default.
- W2000860057 creator A5072079183 @default.
- W2000860057 creator A5074175018 @default.
- W2000860057 creator A5086923762 @default.
- W2000860057 date "2009-07-24" @default.
- W2000860057 modified "2023-10-04" @default.
- W2000860057 title "Cellular basis of uraemic cardiomyopathy: a role for erythropoietin?" @default.
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