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• W2000935302 abstract "Although many tumors express tumor-specific antigens, most fail to stimulate effective immune responses. Tumors generally lack co-stimulatory molecules, which can lead to tolerance of tumor-specific T cells and progressive tumor growth. Here, we demonstrate that the ovalbumin (OVA) transfected EL4 tumor, E.G7-OVA, grows progressively in syngeneic mice even though the tumor can be rejected if the mice are immunized with OVA in adjuvant. E.G7-OVA grew more rapidly in RAG-1 deficient than sufficient mice suggesting that normal mice make an abortive immune response to this tumor. Depletion of gammadelta T cells or IL-10 augmented the ability of B6 mice to reject E.G7-OVA. Spleen cells from normal, but not IL-10 knockout, mice reconstituted rapid tumor growth in gammadelta T cell-deficient mice. Thus, gammadelta T cells play an important role in preventing immune elimination of this tumor by a mechanism that directly or indirectly involves IL-10." @default.
• W2000935302 created "2016-06-24" @default.
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• W2000935302 date "2003-02-01" @default.
• W2000935302 modified "2023-09-25" @default.
• W2000935302 title "Inhibition of tumor rejection by γδ T cells and IL-10" @default.
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• W2000935302 doi "https://doi.org/10.1016/s0008-8749(03)00066-2" @default.
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