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• W2000985786 abstract "The coagulant and inflammatory exacerbation in sepsis is counterbalanced by the protective protein C (PC) pathway. Activated PC (APC) was shown to use the endothelial cell PC receptor (EPCR) as a coreceptor for cleavage of protease activated receptor 1 (PAR1) on endothelial cells. Gene profiling demonstrated that PAR1 signaling could account for all APC-induced protective genes, including the immunomodulatory monocyte chemoattractant protein-1 (MCP-1), which was selectively induced by activation of PAR1, but not PAR2. Thus, the prototypical thrombin receptor is the target for EPCR-dependent APC signaling, suggesting a role for this receptor cascade in protection from sepsis." @default.
• W2000985786 created "2016-06-24" @default.
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• W2000985786 date "2002-06-07" @default.
• W2000985786 modified "2023-10-11" @default.
• W2000985786 title "Activation of Endothelial Cell Protease Activated Receptor 1 by the Protein C Pathway" @default.
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• W2000985786 doi "https://doi.org/10.1126/science.1071699" @default.
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