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- W2001065028 abstract "EVEN BEFORE the neuromuscular blocking effect of certain antibiotics had been discovered, prolonged, 1 and, on occasion, irreversible, 2 respiratory depression was observed after the intraperitoneal administration of relatively large (2- to 5-gm) doses of neomycin sulfate. Subsequent investigation of the neuromuscular effect of various antibiotics on laboratory animals revealed that streptomycin, 3 neomycin sulfate, 4 and certain other 5 antibiotics cause a nondepolarization, or Phase II, 6 block. The neuromuscular block induced by antibiotics is potentiated by ether and antagonized by neostigmine bromide (Prostigmin) and calcium. 3 These observations on the neuromuscular effects of antibiotics were confirmed 7 on human intercostal nervemuscle preparations. The neuromuscular block produced by nondepolarizing relaxants, eg, d-tubocurarine chloride, gallamine (Flaxedil) triethiodide, and antibiotics, is additive. According to the earlier concepts of the mechanism of action of relaxants, it would be expected that antibiotics capable of producing a nondepolarization or Phase II block would" @default.
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- W2001065028 date "1963-02-23" @default.
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- W2001065028 title "Prolonged Respiratory Depression Caused by Drug Combinations" @default.
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- W2001065028 doi "https://doi.org/10.1001/jama.1963.63700080017017e" @default.
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