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• W2001261528 abstract "Temporal restriction of feeding can entrain circadian behavioral and physiological rhythms in mammals. These changes in biological rhythms are postulated to be brought about by a putative food-entrainable oscillator (FEO) that is independent of the suprachiasmatic nucleus (SCN). However, the anatomical substrates and molecular machinery of FEO remain elusive. We report here that mice with a nervous system-specific deletion of Bmal1 , an essential clock component, had a marked deficit in entrainment of locomotor activity by periodic feeding, accompanied by reduced food intake and subsequent loss of body weight. These mice exhibited a nearly normal light-entrainable activity rhythm driven by the SCN, because deletion of the Bmal1 gene in the SCN was only partial. These findings suggest that an SCN-independent FEO in the nervous system requires Bmal1 and plays a critical role in adaptation of circadian locomotor activity and food intake to periodic feeding." @default.
• W2001261528 created "2016-06-24" @default.
• W2001261528 creator A5002913131 @default.
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• W2001261528 date "2011-10-26" @default.
• W2001261528 modified "2023-09-27" @default.
• W2001261528 title "<i>Bmal1</i>in the Nervous System Is Essential for Normal Adaptation of Circadian Locomotor Activity and Food Intake to Periodic Feeding" @default.
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• W2001261528 doi "https://doi.org/10.1523/jneurosci.2801-11.2011" @default.
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