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- W2001399725 endingPage "190" @default.
- W2001399725 startingPage "184" @default.
- W2001399725 abstract "The ability to respond to perturbations in endoplasmic reticulum (ER) function is a fundamentally important property of all cells, but ER stress can also lead to apoptosis. In settings of chronic ER stress, the associated apoptosis may contribute to pathophysiological processes involved in a number of prevalent diseases, including neurodegenerative diseases, diabetes, atherosclerosis and renal disease. The molecular mechanisms linking ER stress to apoptosis are the topic of this review, with emphases on relevance to pathophysiology and integration and complementation among the various apoptotic pathways induced by ER stress." @default.
- W2001399725 created "2016-06-24" @default.
- W2001399725 creator A5077934127 @default.
- W2001399725 creator A5078662102 @default.
- W2001399725 date "2011-03-01" @default.
- W2001399725 modified "2023-10-18" @default.
- W2001399725 title "Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress" @default.
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