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• W2001536413 abstract "목적: 우리는 클로스트리디움 보툴리눔 독소 A형(BoNT/A)이 미페프리스톤으로 유발된 조기진통 쥐 모델에 대해 자궁수축억제 효과가 있는지 in vivo 연구를 실시하였다. 연구방법: 임신 17일, 개복 후 자궁 양측 뿔에 식염수 또는 20 유니트의 BoNT/A를 투여하였다. 임신 18일, 미페프리스톤을 근주하여 자궁의 조기진통을 유발시켰다. 임신 19일, 근전도검사기를 통하여 자궁수축 활동을 측정하였다. 결과: BoNT/A 20 유니트를 투여한 군은 자궁수축 강도가 BoNT/A를 투여하지 않은 군에 비해 45.2% (P＜0.05)로 유의하게 감소하였으며, 자궁수축 기간은 51.6% (P＜0.01)로 유의하게 감소하였다. 하지만 자궁수축 빈도에 대한 감소는 5.6% ( P = 0.4)로 유의하게 감소는 없었다. 결론: 미페프리스톤으로 유발된 조기진통 쥐 모델에서 BoNT/A는 자궁의 수축력을 유의하게 감소시켰다. 자궁수축의 감소는 자궁수축 빈도에 대한 억제효과보다는 자궁수축의 기간 및 강도의 억제효과가 유의했다." @default.
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• W2001536413 date "2012-01-01" @default.
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• W2001536413 title "Clostridium botulinumtoxin A significantly inhibits uterine contractions in rats undergoing mifepristone-induced preterm labor" @default.
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• W2001536413 doi "https://doi.org/10.5468/kjog.2012.55.9.629" @default.
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