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• W2001689142 abstract "Celiac disease (CD) is an enteropathy occurring in genetically susceptible individuals with HLA DQ2 or DQ8 heterodimer caused by ingestion of certain cereals. Until recently it was generally believed that CD is a rare disorder in the United States. More recently, a screening with antiendomysium antibody (EMA) in healthy blood donors in the United States found the prevalence of EMA-positive individuals to be one in 250. The classical form of CD presents with gastrointestinal symptoms between 6 and 18 months of age. Recent studies have shown that almost 50% of patients with newly diagnosed CD do not present with gastrointestinal symptoms The diagnosis of CD can be established when the characteristic changes of the duodenal mucosa are found in a child with signs and/or symptoms consistent with CD, provided that a full and unequivocal clinical remission after withdrawal of gluten is seen, associated with the disappearance of circulating antibodies. Serologic testing for CD has included testing for food protein-directed antibodies (antigliadin, or AGA) and for an autoantibody (EMA). Recently tissue transglutaminase (tTG) was identified as the autoantigen of celiac disease. An enzyme linked immunosorbent assay (ELISA) for immunoglobulin A (IgA) tTG antibodies was found to be 98.1% sensitive and 94.7% specific in patients with biopsy proven CD. While serology should be utilized to screen for CD, particularly in the higher risk populations, intestinal biopsy should be used to confirm the diagnosis. Total lifelong avoidance of gluten ingestion is the cornerstone treatment for the celiac disease." @default.
• W2001689142 created "2016-06-24" @default.
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• W2001689142 date "2002-10-01" @default.
• W2001689142 modified "2023-10-16" @default.
• W2001689142 title "Celiac disease A diagnostic challenge with many facets" @default.
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• W2001689142 doi "https://doi.org/10.1016/s1529-1049(02)00052-1" @default.
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