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- W200179622 abstract "Recently, the Kim Lab has shown that the cystic fibrosis transmembrane conductance regulator (cftr) gene is responsible for mediating resistance to Pseudomonas aeruginosa in a zebrafish infection model. Using the Gene Expression Omnibus, an NCBI functional genomics data repository, it was determined that Smad3, a transcription factor in the TGF-β signaling pathway, is upregulated in the presence of P. aeruginosa. It was found that in our zebrafish model, the Smad3 paralogs Smad3a and Smad3b are upregulated following microinjection of a cftr antisense morpholino oligomer. It was also found that microinjection of Smad3a and Smad3b morpholinos, along with a Smad2 morpholino, and subsequent infection with Pseudomonas aeruginosa resulted in an increase in death, indicating that Smad3 has a protective effect against infection." @default.
- W200179622 created "2016-06-24" @default.
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- W200179622 date "2012-01-01" @default.
- W200179622 modified "2023-09-27" @default.
- W200179622 title "Characterization of Genes in the CFTR-Mediated Innate Immune Response" @default.
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