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• W2001971053 abstract "The cortical EEG of 5 inbred strains of mice susceptible to audiogenic seizures and of 3 inbred strains resistant to them as well as 3 accoustically primed inbred strains was recorded before, during, and after exposure to intense noise once a day for 4 consecutive days beginning at 29 or 30 days of age. None of the resistant mice had a convulsion and all of the susceptible mice had at least one convulsion. Before, during, and after the audiogenic seizure, there was no evidence of spike waves or paroxysmal activity in the trace from the bipolar cortical electrodes. Rather, there might be a slight amplification and acceleration of the trace at the stimulus onset with no further changes during wild circling activity, but with a diminution of the trace during clonic or clonic-tonic convulsions. This pattern was observed for all 5 genetically susceptible strains and for all 3 acoustically primed groups. However, during chemoconvulsive seizures with picrotoxin or thiosemicarbazide, these same mice as well as resistant mice show spike waves and paroxysmal activity of the cortex. It is suggested on the basis of these data that the neural mechanism for the expression of audiogenic seizures and chemoconvulsive seizures is different, that all audiogenic seizures have a common mechanism for expression but not for development of this phenotype, and that the audiogenic seizure is a type of brain stem epilepsy." @default.
• W2001971053 created "2016-06-24" @default.
• W2001971053 creator A5048657712 @default.
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• W2001971053 date "1976-05-01" @default.
• W2001971053 modified "2023-09-23" @default.
• W2001971053 title "Electroencephalographic correlates of the audiogenic seizure response of inbred mice" @default.
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• W2001971053 doi "https://doi.org/10.1016/0031-9384(76)90224-9" @default.
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