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• W2002352252 abstract "Hyperglycemia is a common phenomenon in the early phase of brain injury (BI). The management of blood glucose levels after BI, however, is subject of a growing debate. The occurrence of elevated blood glucose concentrations is linked to increased mortality and worse neurologic outcomes indicating the necessity for therapeutic glucose-lowering. Intensive glucose-lowering therapy, on the other hand, inevitably results in an increased rate of hypoglycemic episodes with detrimental effects on the injured brain. In this review, we give an overview on the current knowledge about causes and pathophysiological consequences of dysglycemia in patients with BI and offer some suggestions for clinical glucose management." @default.
• W2002352252 created "2016-06-24" @default.
• W2002352252 creator A5038506927 @default.
• W2002352252 creator A5064118281 @default.
• W2002352252 date "2014-12-01" @default.
• W2002352252 modified "2023-10-02" @default.
• W2002352252 title "Glycemic control after brain injury: Boon and bane for the brain" @default.
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• W2002352252 doi "https://doi.org/10.1016/j.neuroscience.2014.04.059" @default.
• W2002352252 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24814022" @default.
• W2002352252 hasPublicationYear "2014" @default.
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