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• W2002605819 endingPage "687" @default.
• W2002605819 startingPage "679" @default.
• W2002605819 abstract "The ability of the ocular surface to mount an immune response is in part attributed to a family of proteins called toll-like receptors (TLRs). The latter are evolutionary conserved receptors that recognize and respond to various microbes and endogenous ligands. In addition to their recognition function, TLR activation triggers a complex signal transduction cascade that induces the production of inflammatory cytokines and co-stimulatory molecules, thus initiating innate and adaptive immunity. Toll-like receptor expression at the ocular surface is modulated during infection (e.g. Herpes simplex, bacterial keratitis and fungal keratitis) as well as during various inflammatory conditions (allergic conjunctivitis and dry-eye syndrome). Here recent findings regarding TLR expression and their involvement in various ocular surface diseases are discussed." @default.
• W2002605819 created "2016-06-24" @default.
• W2002605819 creator A5066617821 @default.
• W2002605819 creator A5084515179 @default.
• W2002605819 date "2010-06-01" @default.
• W2002605819 modified "2023-10-09" @default.
• W2002605819 title "Toll-like receptors in ocular surface disease" @default.
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• W2002605819 doi "https://doi.org/10.1016/j.exer.2010.03.012" @default.
• W2002605819 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2905230" @default.

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