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• W2003240244 startingPage "277" @default.
• W2003240244 abstract "Anandamide (arachidonoylethanolamide, AnNH) is shown to activate human platelets, a process which was not inhibited by acetylsalicylic acid (aspirin). Unlike AnNH, hydroperoxides generated thereof by lipoxygenase activity, and the congener (13‐hydroxy)linoleoylethanolamide, were unable to activate platelets, though they counteracted AnNH‐mediated stimulation. On the other hand, palmitoylethanolamide neither activated human platelets nor blocked the AnNH effects. AnNH inactivation by human platelets was afforded by a high‐affinity transporter, which was activated by nitric oxide‐donors up to 225% of the control. The internalized AnNH could thus be hydrolyzed by a fatty acid amide hydrolase (FAAH), characterized here for the first time." @default.
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• W2003240244 date "1999-03-26" @default.
• W2003240244 modified "2023-10-18" @default.
• W2003240244 title "Anandamide activates human platelets through a pathway independent of the arachidonate cascade" @default.
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• W2003240244 doi "https://doi.org/10.1016/s0014-5793(99)00308-7" @default.
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