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• W2003295057 abstract "Pseudomonas aeruginosa infection, one of the major complications of burn wounds, may lead to sepsis and death. Using the Multi-Probe Template/RNase protection assay, we have compared the expression of different cytokine genes within the skin and livers of thermally injured mice infected with P. aeruginosa PAO1. Thermal injury alone enhanced or up-regulated certain cytokines, including macrophage colony-stimulating factor (M-CSF), interleukin 1 (IL-1)RI, IL-1β, macrophage inflammatory protein (MIP)-1β and MIP-2; while PAO1 challenge alone up-regulated tumour necrosis factor α (TNF-α) and transforming growth factor β (TGF-β) expression. The combination of thermal injury plus PAO1 infection enhanced the expression of several pro-inflammatory and haematopoietic cytokines [stem cell factor (SCF), leukocyte inhibitory factor (LIF), IL-6 and TNF-α]; induced the expression of granulocyte–macrophage colony-stimulating factor (GM-CSF) and G-CSF by 5 h and the expression of additional cytokines, including TGF-β, TNF-β, lymphotoxin β (LT-β), interferon γ (IFN-γ), and IFN-β by 40 h post-burn/infection. While the most intense cytokine expression occurred in the skin, the majority of cytokines tested were also expressed in the liver by 40 h post-burn/infection. These results suggest that in P. aeruginosa infection of burn wounds: (1) up-regulation of the expression of different cytokines, locally and within the livers of burned mice, is an indication of P. aeruginosa -induced sepsis; and (2) IL-6 and G-CSF play an important role in the host response mechanism." @default.
• W2003295057 created "2016-06-24" @default.
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• W2003295057 date "2001-11-01" @default.
• W2003295057 modified "2023-10-10" @default.
• W2003295057 title "THE EFFECTS OF INFECTION OF THERMAL INJURY BY PSEUDOMONAS AERUGINOSA PAO1 ON THE MURINE CYTOKINE RESPONSE" @default.
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• W2003295057 doi "https://doi.org/10.1006/cyto.2001.0960" @default.
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