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- W2003493239 abstract "Use of nonsteroidal anti-inflammatory drugs has been shown to result in a 40% to 50% reduction in the relative risk of developing colorectal cancer. Cyclooxygenase-2 (COX-2) overexpression occurs in 43% of human invasive breast cancers and 63% of ductal carcinomas in situ. There is considerable in vitro, animal model, and epidemiologic evidence to suggest that COX-2 may play some role in breast tumor initiation and progression. PGE2 is a major downstream mediator of COX-2 that promotes cellular proliferation and angiogenesis, makes cells resistant to apoptosis, enhances invasiveness, and modulates immunosuppression. COX-2 and COX-2-derived PGE2 may be involved in mammary carcinogenesis. Therefore, COX-2 selective inhibitors may have a role in breast cancer prevention." @default.
- W2003493239 created "2016-06-24" @default.
- W2003493239 creator A5014835615 @default.
- W2003493239 creator A5090373031 @default.
- W2003493239 date "2004-02-01" @default.
- W2003493239 modified "2023-10-16" @default.
- W2003493239 title "Cyclooxygenase-2: a potential target in breast cancer" @default.
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- W2003493239 doi "https://doi.org/10.1053/j.seminoncol.2004.01.008" @default.
- W2003493239 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/15052544" @default.
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