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• W2003670956 endingPage "905" @default.
• W2003670956 startingPage "901" @default.
• W2003670956 abstract "Autoimmune bullous diseases (AIBDs) are characterized by blisters and erosions on the skin and/or mucous membranes, which are caused by autoantibodies directed to structural proteins of the epidermis and the epidermal basement membrane zone. This Viewpoint Essay discusses the contribution by autoreactive T cells to the pathogenesis of bullous pemphigoid, pemphigus and epidermolysis bullosa acquisita, with an emphasis on studies using active animal mouse models for these diseases. Previous studies have demonstrated that cytokines produced by autoreactive T cells, the interaction between antigen-specific T cells and B cells and the function of regulatory T cells are likely related to the pathogenesis of AIBDs. In interpreting the experimental results, the limitations of those animal models should be considered. Further understanding of the pathogenicity of autoreactive CD4(+) T cells may lead to disease-specific treatments." @default.
• W2003670956 created "2016-06-24" @default.
• W2003670956 creator A5013596644 @default.
• W2003670956 creator A5067879929 @default.
• W2003670956 date "2012-09-28" @default.
• W2003670956 modified "2023-09-27" @default.
• W2003670956 title "Evidence for pathogenicity of autoreactive T cells in autoimmune bullous diseases shown by animal disease models" @default.
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• W2003670956 doi "https://doi.org/10.1111/exd.12011" @default.
• W2003670956 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23016514" @default.
• W2003670956 hasPublicationYear "2012" @default.
• W2003670956 type Work @default.

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