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• W2003752465 abstract "Hans-Gerhard Creutzfeldt and Alfons Jakob independently authored clinical and pathologic descriptions of a new syndrome in the 1920s. This syndrome, which subsequently came to be named after them, was characterized by dementia, motor and coordination abnormalities, a fatal course, and pathologic findings of diffuse spongiform neuronal degeneration. Although it appeared for many years to be little more than a medical curiosity, Creutzfeldt-Jakob disease attained widespread attention by its pathologic similarity to kuru and bovine spongiform encephalopathy, “mad cow disease.” Because there are sporadic, familial, and iatrogenic forms of Creutzfeldt-Jakob disease, it is considered to have both genetic and infectious aspects. Although its causation has for some time been ascribed to “slow viruses,” the etiology of Creutzfeldt-Jakob disease is currently thought to be due to prions, small proteinaceous infectious particles that have genetic encoding. The debate regarding whether the appearance of atypical Creutzfeldt-Jakob disease can be linked to the epidemic of “mad cow disease” is currently unresolved." @default.
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• W2003752465 date "1997-09-01" @default.
• W2003752465 modified "2023-09-29" @default.
• W2003752465 title "From Creutzfeldt-Jakob disease to the mad cow epidemic" @default.
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• W2003752465 doi "https://doi.org/10.1016/s0736-4679(97)00152-2" @default.
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