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- W2003805940 abstract "Brain edema in acute liver failure (ALF) remains lethal. Cytotoxic mechanisms associated with brain edema have been well recognized, but the role of vasogenic mechanisms of brain edema has not been explored. Intact tight junctions (TJs) between brain capillary endothelial cells are critical for normal BBB function. Recent reports found significant alterations in the tight junction elements including occludin and claudin-5, suggesting a vasogenic injury in the blood-brain barrier (BBB) integrity. However, the role of TJ in ALF has not been completely understood. This paper reviews the role of the paracellular tight junction in the increased selective BBB permeability that leads to brain edema in ALF and furthermore explores the effect of systemic inflammatory cytokines on the tight junction dysfunction." @default.
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- W2003805940 date "2013-01-01" @default.
- W2003805940 modified "2023-10-17" @default.
- W2003805940 title "Alterations of Blood-Brain Barrier and Associated Factors in Acute Liver Failure" @default.
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- W2003805940 doi "https://doi.org/10.1155/2013/841707" @default.
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