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- W2003882961 endingPage "148" @default.
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- W2003882961 abstract "Sepsis is a clinical syndrome that is usually induced by bacterial infections. It is generally assumed that the syndrome results from an excessive triggering of endogenous inflammatory mediators by the invading microorganisms. These mediators include substances released by activated monouytes, macrophages, endothelial cells and neutrophils such as cytokines, reactive oxygen species and proteases, as well as activation products of coagulation, fibrinolysis, contact and complement systems. Recent studies have suggested that cytokines and complement activation products may have overlapping biological activities. In addition, multiple interactions in vitro as well as in vivo between cytokines and complement have been described. Here we will review some of these recent studies and will discuss their relevance for the pathogenesis of sepsis and septic shock." @default.
- W2003882961 created "2016-06-24" @default.
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- W2003882961 creator A5080558143 @default.
- W2003882961 date "1992-09-01" @default.
- W2003882961 modified "2023-10-18" @default.
- W2003882961 title "Interplay of complement and cytokines in the pathogenesis of septic shock" @default.
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