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- W2003968045 endingPage "497" @default.
- W2003968045 startingPage "485" @default.
- W2003968045 abstract "Platelet function is a balance between factors determined at thrombopoiesis and prothrombotic and antithrombotic mediators from the vascular wall. Nitric oxide is a crucial vasodilator and inhibitor of platelet activation synthesized constitutively by the vascular endothelium. In some pathological states this synthesis may be impaired leading to a prothrombotic state. In other situations, synthesis may be increased by a second inducible enzyme. Nitric oxide synergizes with other antithrombotic vasodilators such as prostacyclin and is opposed by prothrombotic vasoconstrictor mediators such as thromboxane. Platelets are anucleate and their reactivity is partly determined at thrombopoiesis by their progenitor cell, the megakaryocyte. In thrombotic states, such as myocardial infarction, larger, more reactive platelets from larger megakaryocytes are observed." @default.
- W2003968045 created "2016-06-24" @default.
- W2003968045 creator A5027251851 @default.
- W2003968045 creator A5030238742 @default.
- W2003968045 date "1994-09-01" @default.
- W2003968045 modified "2023-10-16" @default.
- W2003968045 title "4 Endogenous mediators and thrombophilia" @default.
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