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• W2004158223 abstract "We have previously shown that young guinea pigs repeatedly exposed to sidestream cigarette smoke (SS) develop decreased airway reactivity of the C-fiber system without changing reactivity to one of its neurotransmitters, substance P (SP). This study was designed to determine whether the decreased reactivity was due to decreased responsiveness to another neurotransmitter, neurokinin A (NKA), decreased lung SP content, decreased affinity or number of NK1 receptors, and/or decreased number of C-fibers. Duncan Hartley guinea pigs were exposed to filtered air (FA) or to SS for 6 h/day, 5 days/week for 5 weeks starting at 1 week of age. SS exposure did not change, (1) airway reactivity to NKA injected into the pulmonary artery of their isolated perfused lungs (n = 6–7 each group), (2) lung SP content as measured by enzyme immunoassay (n = 12 each group), (3) NK, receptor number or affinity as measured by radioligand binding (n = 7 each group), or (4) SP-immunoreactive nerve profiles of the terminal bronchioles or small airways (n = 6 each group). Thus, SS exposure does not decrease C-fiber system by reducing NKA responsiveness, decreasing SP content, changing NK1 receptors, or decreasing the number of C-fibers." @default.
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• W2004158223 date "1996-09-02" @default.
• W2004158223 modified "2023-09-26" @default.
• W2004158223 title "Effects of extended sidestream smoke exposure on components of the C-fiber axon reflex" @default.
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• W2004158223 doi "https://doi.org/10.1016/0300-483x(96)03403-8" @default.
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