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• W2004361024 abstract "Lethargic (lh/lh) mice, which function as an animal model of absence seizures, have spontaneous seizures that have behavioral and electrographic features and anticonvulsant sensitivity similar to those of human absence seizures. Antagonists of the γ-aminobutyric acidB (GABAB) receptor suppressed these seizures in lethargic mice, whereas agonists of GABAB receptors exacerbated them. Furthermore, GABAB receptor binding and synaptically evoked GABAB receptor-mediated inhibition of N-methyl-D-aspartate responses were selectively increased in lh/lh mice. Therefore, enhanced GABAB receptor-mediated synaptic responses may underlie absence seizures in lh/lh mice, and GABAB receptor antagonists hold promise as anticonvulsants for absence seizures." @default.
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• W2004361024 date "1992-07-17" @default.
• W2004361024 modified "2023-09-26" @default.
• W2004361024 title "The Role of GABA _B Receptor Activation in Absence Seizures of Lethargic ( <i>lh</i> / <i>lh</i> ) Mice" @default.
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• W2004361024 doi "https://doi.org/10.1126/science.1321503" @default.
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