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- W2005046746 endingPage "59" @default.
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- W2005046746 abstract "Nonalcoholic fatty liver disease (NAFLD) is a consequence of insulin resistance encompassing a spectrum that extends from simple hepatic steatosis through to nonalcoholic steatohepatitis (NASH), a condition that may progress to cirrhosis with its associated complications. A subset of nuclear receptors act as intracellular sensors for cholesterol metabolites, free fatty acids, and a range of other lipophilic molecules with pivotal roles in energy homeostasis and inflammation. These receptors represent attractive drug targets for the management of NAFLD and NASH as well as related conditions such as type 2 diabetes and the broader metabolic syndrome. To date, human studies have concentrated on peroxisome proliferator-activated receptor (PPAR) agonists, particularly those directed at PPARγ. However, these drugs have significant limitations, so alternate approaches to nuclear receptor targeting are being explored." @default.
- W2005046746 created "2016-06-24" @default.
- W2005046746 creator A5011865871 @default.
- W2005046746 creator A5049828619 @default.
- W2005046746 date "2007-12-12" @default.
- W2005046746 modified "2023-09-23" @default.
- W2005046746 title "Nonalcoholic Fatty Liver Disease: Pathogenesis and Potential for Nuclear Receptors as Therapeutic Targets" @default.
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