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- W2005149728 abstract "There is accumulating evidence showing that lupus anticoagulants (LA) are more strongly associated with thrombosis than anticardiolipin antibodies. In addition, indirect evidence has been presented indicating that β2GPI-dependent LA are more strongly associated with thrombosis than prothrombin-dependent LA. From this, one may assume that anti-β2GPl antibodies with LA activity are more pathogenic than anti-β2GPI antibodies without LA activity. Therefore, it is of the utmost importance to understand the molecular basis on which some anti-β2GPI antibodies behave as LA. In this presentation, the current knowledge on the interaction of β2GPI with phospholipids and with anti-β2GPI antibodies is reviewed and an integrated model for the anti-β2GPI - dependent LA activity is proposed with implications for a pathogenic role of these particular antibodies." @default.
- W2005149728 created "2016-06-24" @default.
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- W2005149728 date "1998-02-01" @default.
- W2005149728 modified "2023-10-18" @default.
- W2005149728 title "Mechanism of action of β2-glycoprotein I-dependent lupus anticoagulants" @default.
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- W2005149728 doi "https://doi.org/10.1177/096120339800700206" @default.
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