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- W2005168293 abstract "Oxidation and glycation of lipoproteins increases its atherogenicity. Elevated levels of glycated low-density lipoprotein (gly-LDL) are associated with increased cardiovascular risk in diabetes. Previous studies have demonstrated that nitric oxide acts as a vasoprotective molecule and endothelial nitric oxide synthase (eNOS) is responsible for its production and endothelium-dependent vasodilation. Endothelial dysfunction is associated with diabetic cardiovascular complications. However, the relationships between endoplasmic reticulum (ER), oxidative stress and eNOS activity inhibited by gly-LDL largely remains unknown." @default.
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- W2005168293 date "2014-10-01" @default.
- W2005168293 modified "2023-09-26" @default.
- W2005168293 title "Effect of Glycated Low-Density Lipoprotein on Endothelial Nitric Oxide Synthase Content in Vascular Endothelial Cells: Involvement of Transmembrane Signalling and Endoplasmic Reticulum Stress" @default.
- W2005168293 doi "https://doi.org/10.1016/j.jcjd.2014.07.035" @default.
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