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- W2005368419 abstract "Abstract There are P cells in the human and canine AV (atrioventricular) node which are virtually devoid of gap junctions. All other components of myocardial cellular connections are calcium-dependent except the gap junction. Direct perfusion of disodium EDTA through the AV node artery of 16 anaesthetized dogs produced three immediate effects: complete AV block, a rapid irregular atrial rhythm and a separate rapid irregular ventricular rhythm. The atrial arrhythmia was short in duration and sinus rhythm resumed, initially with complete AV and VA block; both waned until normal AV conduction returned in each dog. In 3 of the 16 dogs there was transient complete AV block during which two independent His potentials were separately associated with the atrial and ventricular complexes. When conducted sinus rhythm resumed, there was initially A-H prolongation (but not H-V). Atropine, propranolol and reserpine had no influence on any electrophysiologic effect of EDTA. Both tachycardias probably originate in P cells of the AV node, the irregularity being attributable to varying enhancement of automaticity plus functional disaggregation of P cells. AV block is attributed to failure of conduction between disaggregated P cells, which in turn must be an obligatory pathway for normal AV conduction, because of their anatomic interposition. The findings further suggest that the AV nodal P cells are the site of the normal 40 ms delay in AV conduction, and that they may be the site of origin of the His potential." @default.
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- W2005368419 date "1976-05-01" @default.
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- W2005368419 title "Selective experimental chelation of calcium in the AV node and His bundle" @default.
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- W2005368419 doi "https://doi.org/10.1016/0022-2828(76)90041-9" @default.
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