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- W2005382336 abstract "Slow axonal transport is a multivariate phenomenon implicated in several neurodegenerative disorders. Recent reports have unraveled the molecular basis of the transport of certain slow component proteins, such as the neurofilament subunits, tubulin, and certain soluble enzymes such as Ca2+/calmodulin-dependent protein kinase IIa (CaM kinase IIa), etc., in tissue cultured neurons. In addition, genetic analyses also implicate microtubule-dependent motors and other housekeeping proteins in this process. However, the biological relevance of this phenomenon is not so well understood. Here, the authors have discussed the possibility of adopting neurogenetic analyses in multiple model organisms to correlate molecular level measurements of the slow transport phenomenon to animal behavior, thus facilitating the investigation of its biological efficacy." @default.
- W2005382336 created "2016-06-24" @default.
- W2005382336 creator A5021063183 @default.
- W2005382336 creator A5077599963 @default.
- W2005382336 date "2012-07-27" @default.
- W2005382336 modified "2023-10-16" @default.
- W2005382336 title "Neurogenetics of Slow Axonal Transport: From Cells to Animals" @default.
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- W2005382336 doi "https://doi.org/10.3109/01677063.2012.699564" @default.
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