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• W2005406876 startingPage "133" @default.
• W2005406876 abstract "p63 is the most recently discovered but most ancient member of the p53 family. In marked contrast to p53, p63 is highly expressed in embryonic ectoderm and in the basal, regenerative layers of many epithelial tissues in the adult. The p63-knockout mouse dies at birth and lacks limbs, epidermis, prostate, breast and urothelial tissues, apparently owing to the loss of stem cells required for these tissues. Significantly, several dominant human syndromes involving limb development and/or ectodermal dysplasia have been mapped to chromosome 3q27 and ultimately the gene encoding p63. The heterozygous p63mutations are distinct for each of the syndromes and are thought to act through both dominant-negative and gain-of-function mechanisms rather than a loss-of-function haploinsufficiency. The allele specificity of these syndromes offers unique molecular insights into the poorly understood actions of p63 in limb development, ectodermal-mesodermal interactions and stem cell maintenance." @default.
• W2005406876 created "2016-06-24" @default.
• W2005406876 creator A5013649683 @default.
• W2005406876 creator A5066748955 @default.
• W2005406876 date "2002-03-01" @default.
• W2005406876 modified "2023-09-26" @default.
• W2005406876 title "Mutations in the p53 homolog p63: allele-specific developmental syndromes in humans" @default.
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• W2005406876 doi "https://doi.org/10.1016/s1471-4914(01)02260-2" @default.
• W2005406876 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11879774" @default.
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