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- W2005672017 abstract "Cyclooxygenases and their metabolites are important regulators of inflammatory responses and play critical roles in regulating the differentiation of T helper cell subsets in inflammatory diseases. In this review, we highlight new information on regulation of T helper cell subsets by cyclooxygenases and their metabolites. Prostanoids influence cytokine production by both antigen presenting cells and T cells to regulate the differentiation of naïve CD4+ T cells to Th1, Th2 and Th17 cell phenotypes. Cyclooxygenases and PGE2 generally exacerbate Th2 and Th17 phenotypes, while suppressing Th1 differentiation. Thus, cycloxygenases may play a critical role in diseases that involve immune cell dysfunction. Targeting of cyclooxygenases and their eicosanoid products may represent a new approach for treatment of inflammatory diseases, tumors and autoimmune disorders." @default.
- W2005672017 created "2016-06-24" @default.
- W2005672017 creator A5000941083 @default.
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- W2005672017 creator A5057235990 @default.
- W2005672017 creator A5061139170 @default.
- W2005672017 creator A5073886839 @default.
- W2005672017 date "2013-07-01" @default.
- W2005672017 modified "2023-10-02" @default.
- W2005672017 title "Regulation of T helper cell subsets by cyclooxygenases and their metabolites" @default.
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