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- W2006111149 abstract "Using a mouse mutagenesis screen, we have identified CD83 as being critical for the development of CD4<sup>+</sup> T cells and for their function postactivation. CD11c<sup>+</sup> dendritic cells develop and function normally in mice with a mutated <i>CD83</i> gene but CD4<sup>+</sup> T cell development is substantially reduced. Additionally, we now show that those CD4<sup>+</sup> cells that develop in a CD83 mutant animal fail to respond normally following allogeneic stimulation. This is at least in part due to an altered cytokine expression pattern characterized by an increased production of IL-4 and IL-10 and diminished IL-2 production. Thus, in addition to its role in selection of CD4<sup>+</sup> T cells, absence of CD83 results in the generation of cells with an altered activation and cytokine profile." @default.
- W2006111149 created "2016-06-24" @default.
- W2006111149 creator A5074983628 @default.
- W2006111149 date "2004-02-12" @default.
- W2006111149 modified "2023-09-26" @default.
- W2006111149 title "Gulf war syndrome case collapses" @default.
- W2006111149 doi "https://doi.org/10.1136/bmj.328.7436.367-b" @default.
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