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- W2006162854 abstract "This study examined whether alterations in intracellular or extracellular Ca2+ mobilization were related to differences in caffeine and phenylephrine (PHE)-induced contractions between two-kidney, one-clip hypertensive (2K-1C) and normotensive (2K) rat aortas. After depletion and reloading of intracellular Ca2+ stores, caffeine and PHE-induced contractions in Ca2+-free solution were increased in 2K-1C. Thapsigargin reduced the contraction to caffeine in 2K-1C and 2K with similar sensitivity. PHE-induced contraction in 1.6-mM Ca2+ solution was decreased in 2K-1C, and nifedipine was less effective in lowering this response. The responsiveness to extracellular Ca2+ was decreased in 2K-1C hypertensive rat aortas. Our results indicate an increased intracellular Ca2+ stores that are not related to alteration in Ca2+-ATPase function and a lower contribution of L-type channels to the contraction of 2K-1C aortas." @default.
- W2006162854 created "2016-06-24" @default.
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- W2006162854 date "1999-12-01" @default.
- W2006162854 modified "2023-10-14" @default.
- W2006162854 title "Contribution of sarcoplasmic reticulum calcium uptake and L-type calcium channels to altered vascular responsiveness in the aorta of renal hypertensive rats" @default.
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- W2006162854 doi "https://doi.org/10.1016/s0306-3623(99)00042-7" @default.
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