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- W2006207287 abstract "Dear SirA 58-year-old female had symmetric spasticity,hyperreflexia, extensor plantar responses, spasticdysarthria, and pseudobulbar symptoms. The uppermotor neuron involvement had progressed graduallyover more than five years, leading to weakness in allfour limbs and pseudobulbar symptoms, requiring apercutaneous endoscopic gastrostomy. Denervationpotentials were not detectable on needle electro-myography. Primary lateral sclerosis was diagnosedclinically. Cranial magnetic resonance imagingshowed hyperintensity along the corticospinal tracts(CST) on fluid-attenuated inversion recovery(FLAIR) (Figure 1A) and T2-weighted images,with high signal intensity in the CST on diffusion-weighted imaging (DWI) (Figure 1B).High signal intensity was evident in the CST onDWI, extending from the precentral gyrus andpassing through the centrum semiovale, periventri-cular white matter, internal capsule, and brainstem.A single case of amyotrophic lateral sclerosis inwhich motor tract degeneration was highlightedmildly on DWI of recent lesions has been documen-ted (1). FLAIR images showed that abnormalitiesextended beyond the CST and were therefore notdiagnostic (1), whereas DWI images in our patientshowed abnormalities restricted to the CST. Becausechanges on DWI may be related to T2 abnormalities(‘T2 shine-through’), it would have been helpful ifsome FLAIR images had been obtained at the samelevels as the DWI images, thereby confirming uppermotor neuron disease.Reference" @default.
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- W2006207287 date "2009-01-01" @default.
- W2006207287 modified "2023-10-16" @default.
- W2006207287 title "Upper motor neuron involvement and conventional diffusion weighted imaging" @default.
- W2006207287 cites W2092210583 @default.
- W2006207287 doi "https://doi.org/10.1080/17482960903039929" @default.
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