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• W2006231708 abstract "Penile erection is a neurovascular process controlled by numerous tightly regulated events, and occurs in response to the activation of pro-erectile autonomic pathways. It is dependent on an adequate inflow of blood to the erectile tissue through both endothelium-dependent vasodilatation and corporal smooth muscle relaxation. Pathologic alteration in the endothelium of penile vasculature and/or erectile tissue and/or impairment of neurovascular processes can result in erectile dysfunction (ED). Both cardiovascular disease (CVD) and ED have been linked to endothelial dysfunction. Endothelial dysfunction is a vascular condition resulting in a diminished vasodilatory response to pharmacologic and physiologic stressors. Endothelial dysfunction may be a pathophysiologic mechanism underlying both ED and CVD, forming a unifying link between these two conditions. Furthermore, in the general population and in men with diabetes or obesity, ED may be a valuable early marker for serious subclinical CVD, coronary artery disease and atherosclerosis." @default.
• W2006231708 created "2016-06-24" @default.
• W2006231708 creator A5019920991 @default.
• W2006231708 date "2008-11-10" @default.
• W2006231708 modified "2023-10-04" @default.
• W2006231708 title "New horizons in erectile and endothelial dysfunction research and therapies" @default.
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• W2006231708 doi "https://doi.org/10.1038/ijir.2008.46" @default.
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